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Consistent with this, glutaminolysis-derived -KG promotes M2 activation by increasing fatty acid oxidation and Jmjd3-dependent epigenetic reprogramming of M2-related genes 142

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However, the complex tumor microenvironment of HGSOC presents challenges for the clinical application of vaccines, with tumor-associated macrophages and other immunosuppressive cells potentially inhibiting vaccine-induced immune responses (136)